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For instance, in lung cancer cell models, the ability to re-enter the cell cycle is specific to a distinct fraction of DTPs, with cycling and non-cycling DTPs displaying distinct transcriptional and metabolic programmes that cluster in specific cell lineages 11

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It does not directly stimulate fibroblast proliferation under normal culture conditions, but substantially increases fibroblast survival under hydrogen peroxide-induced oxidative stress — acting as a cytoprotective enhancer of the endogenous repair response rather than a direct mitogenic agent