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Although TrkB.T1 was found to be involved in the pathogenesis of PD through the negative regulation of BDNF/TrkB signaling, the role of other TrkB isoforms is not fully understood
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In the context of atherosclerosis, the regulatory effects of GA on foam cells and macrophages require more detailed investigation, especially as the specific mechanisms of its anti-ASCVD actions have not been fully elucidated
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